Mitochondrial dysfunction and associated enhanced ROS are connected to aging, and specifically to senescence and inflammation. The maintenance of mitochondrial activity is a prime therapeutic objective as murine scientific studies have demonstrated beneficial effects of PGC1α overexpression on many aging-suitable biological processes which includes metabolism and ROS Handle(Dillon et al. https://sa-ekim18516.slypage.com/28790505/mezoterapi-cilt-an-overview